p17 Under lamellar body count, should read "blood contamination increases the LBC count, and meconium decreases it"
p 154 the most commonly gene affected is ankyrin; whereas in the Companion on p166 the answer to the most commonly affected gene is spectrin (the answer should be ankyrin)
p 28 (column I) TSH paragraph reads "TSH is elevated in true hypothyroidism and elevated in hypothyroidism"
p 154 stomatocytic type of HE confers resistance to P falciprum and P vivax.
☀: Da Costa L, et al, Hereditary spherocytosis, elliptocytosis, and other red cell membrane disorders, Blood Rev (2013), http://dx.doi.org/10.1016/j.blre.2013.04.003
In the Companion question book, p. 74: the answer explanation directly contradicts the stated correct answer regarding weak D etiology. According to the Compendium p 65, weak D is a normal antigen in low quantities and partial D is an abnormal antigen in normal quantities. Both of these support the assumption that the answer to question 62 cannot be E as printed.
p 219, under vWD type 2a, the Compendium lists the molecular defect as being in exon 28. However, exon 28 mutations are more specific to types 2M and 2b than 2a, although they do account for part of 2a cases.
James, PD, Goodeve, AC, von Willebrand Disease. Genetics in Medicine. 13 365-376, 2011. doi:10.1097/GIM.0b013e3182035931